ARDMS ECHO Study Guide 2026
Everything you need to pass the ARDMS ECHO exam in one place: the exam format, every topic to study, real practice questions with explanations, flashcards, and full-length practice tests. Free, no sign-up needed.
📋 ARDMS ECHO Exam Format at a Glance
📚 ARDMS ECHO Topics to Study (34)
✍️ Sample ARDMS ECHO Questions & Answers
1. The RVOT acceleration time (AT) is used to estimate mean pulmonary artery pressure. Which formula applies?
mPAP ≈ 79 − (0.45 × RVOT AT in ms) is a validated formula; shorter AT corresponds to higher pulmonary artery pressure.
2. What is the advantage of using transesophageal echocardiography (TEE) over transthoracic echocardiography (TTE)?
Transesophageal echocardiography (TEE) offers a significant advantage over transthoracic echocardiography (TTE) by providing superior image quality, particularly for posterior cardiac structures. Placing the transducer in the esophagus positions it much closer to the heart, bypassing the ribs, lungs, and chest wall. This proximity and lack of acoustic impedance result in higher resolution images, which are crucial for evaluating structures like the left atrium, mitral valve, and aorta.
3. What does the velocity-time integral (VTI) of the LVOT represent?
LVOT VTI represents the stroke distance — the column of blood ejected per heartbeat — and when multiplied by LVOT CSA gives stroke volume.
4. What is the most common etiology of isolated severe tricuspid regurgitation in the United States?
Functional TR caused by RV dilation and tricuspid annular dilation (from pulmonary hypertension or LV dysfunction) is the most common cause of severe TR in the US.
5. Which parameter best differentiates severe mitral regurgitation from moderate on echocardiography using the PISA method?
Severe primary MR criteria include EROA ≥0.40 cm² and regurgitant volume ≥60 mL per ASE/AHA/ACC guidelines.
6. A ventricular septal defect (VSD) with left-to-right shunt produces which hemodynamic finding on echo?
Left-to-right VSD shunting causes increased pulmonary flow returning to the left heart, resulting in LV and LA volume overload.