Shock Management and Resuscitation Flashcards
6 cards from real FP-C practice questions. Tap to flip, then mark Knew It or Still Learning — missed cards come back until you master them.
Read the first 6 Shock Management and Resuscitation flashcards as text
A hypotensive patient is diagnosed with tension pneumothorax. After needle decompression, the pressure is relieved but hypotension persists. What is the most likely explanation?
Answer: Concurrent hemorrhagic shock from associated injuries
Persistent hypotension after successful tension pneumothorax decompression should prompt evaluation for concurrent hemorrhage or other causes of obstructive/hypovolemic shock.
What is the primary mechanism by which tranexamic acid (TXA) improves outcomes in hemorrhagic shock?
Answer: Inhibits fibrinolysis by blocking plasminogen activation
Tranexamic acid competitively inhibits plasminogen activation, preventing clot breakdown (fibrinolysis) and preserving existing thrombus in hemorrhagic wounds.
A patient in septic shock remains hypotensive after norepinephrine uptitration. Which second-line vasopressor is recommended to spare catecholamine dosing?
Answer: Vasopressin 0.03-0.04 units/min
Vasopressin at 0.03-0.04 units/min is the standard add-on vasopressor for refractory septic shock, acting via V1 receptors independently of adrenergic pathways.
Which end-point best indicates adequate resuscitation has been achieved in a septic shock patient during transport?
Answer: Lactate clearance ≥10% and urine output ≥0.5 mL/kg/hr
Lactate clearance of at least 10% combined with adequate urine output indicates improved tissue perfusion and is a validated resuscitation endpoint in sepsis.
In hemorrhagic shock, what is the maximum crystalloid volume recommended before transitioning to blood products per damage control resuscitation principles?
Answer: 1-1.5 L
Damage control resuscitation limits crystalloid to 1-1.5 L before transitioning to blood products to avoid dilutional coagulopathy and excessive interstitial edema.
Which type of shock is MOST likely in a patient with a history of recent myocardial infarction who now presents with respiratory distress, bilateral crackles, S3 gallop, and hypotension?
Answer: Cardiogenic shock
Post-MI presentation with pulmonary edema, S3 gallop, and hypotension is classic for cardiogenic shock from left ventricular pump failure.