Cardiovascular Emergencies Flashcards
7 cards from real CCRN practice questions. Tap to flip, then mark Knew It or Still Learning โ missed cards come back until you master them.
Read the first 7 Cardiovascular Emergencies flashcards as text
A patient in cardiogenic shock has cold extremities, hypotension, and pulmonary congestion. Which hemodynamic profile is expected?
Answer: Low CO, high PCWP, high SVR
Cardiogenic shock features low cardiac output, elevated filling pressures (PCWP), and compensatory high systemic vascular resistance.
Which medication is a first-line inotrope for a patient in cardiogenic shock with adequate blood pressure?
Answer: Dobutamine
Dobutamine improves contractility and cardiac output and is preferred when blood pressure is marginally adequate.
How does an intra-aortic balloon pump improve cardiac function in cardiogenic shock?
Answer: It increases coronary perfusion during diastole and reduces afterload during systole
The IABP inflates in diastole to augment coronary perfusion and deflates in systole to reduce afterload.
A patient with acute decompensated heart failure has an SpO2 of 84% and crackles throughout. What is the priority intervention?
Answer: Provide supplemental oxygen and consider noninvasive positive pressure ventilation
Oxygenation and positive pressure ventilation improve gas exchange and reduce preload in acute pulmonary edema.
Which sign distinguishes right-sided heart failure from left-sided heart failure?
Answer: Jugular venous distention and peripheral edema
Right-sided failure causes systemic venous congestion with JVD and peripheral edema, whereas left-sided failure causes pulmonary symptoms.
A heart failure patient's BNP is markedly elevated. What does this indicate?
Answer: Increased ventricular wall stretch and volume overload
BNP is released in response to ventricular stretch, so elevation reflects volume overload and heart failure severity.
Why are ACE inhibitors a cornerstone of chronic heart failure management?
Answer: They reduce afterload and prevent ventricular remodeling
ACE inhibitors lower afterload and inhibit the RAAS, slowing maladaptive ventricular remodeling.