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Cardiovascular Emergencies Flashcards

7 cards from real CCRN practice questions. Tap to flip, then mark Knew It or Still Learning โ€” missed cards come back until you master them.

Read the first 7 Cardiovascular Emergencies flashcards as text
  1. A patient in cardiogenic shock has cold extremities, hypotension, and pulmonary congestion. Which hemodynamic profile is expected?

    Answer: Low CO, high PCWP, high SVR

    Cardiogenic shock features low cardiac output, elevated filling pressures (PCWP), and compensatory high systemic vascular resistance.

  2. Which medication is a first-line inotrope for a patient in cardiogenic shock with adequate blood pressure?

    Answer: Dobutamine

    Dobutamine improves contractility and cardiac output and is preferred when blood pressure is marginally adequate.

  3. How does an intra-aortic balloon pump improve cardiac function in cardiogenic shock?

    Answer: It increases coronary perfusion during diastole and reduces afterload during systole

    The IABP inflates in diastole to augment coronary perfusion and deflates in systole to reduce afterload.

  4. A patient with acute decompensated heart failure has an SpO2 of 84% and crackles throughout. What is the priority intervention?

    Answer: Provide supplemental oxygen and consider noninvasive positive pressure ventilation

    Oxygenation and positive pressure ventilation improve gas exchange and reduce preload in acute pulmonary edema.

  5. Which sign distinguishes right-sided heart failure from left-sided heart failure?

    Answer: Jugular venous distention and peripheral edema

    Right-sided failure causes systemic venous congestion with JVD and peripheral edema, whereas left-sided failure causes pulmonary symptoms.

  6. A heart failure patient's BNP is markedly elevated. What does this indicate?

    Answer: Increased ventricular wall stretch and volume overload

    BNP is released in response to ventricular stretch, so elevation reflects volume overload and heart failure severity.

  7. Why are ACE inhibitors a cornerstone of chronic heart failure management?

    Answer: They reduce afterload and prevent ventricular remodeling

    ACE inhibitors lower afterload and inhibit the RAAS, slowing maladaptive ventricular remodeling.