Reaccreditation and Maintenance Flashcards
7 cards from real ABP practice questions. Tap to flip, then mark Knew It or Still Learning — missed cards come back until you master them.
Read the first 7 Reaccreditation and Maintenance flashcards as text
Under ABP reaccreditation requirements, board-certified periodontists must recertify every how many years?
Answer: 10 years
The ABP requires diplomates to recertify every 10 years to maintain board certification status.
Which clinical finding during a supportive periodontal therapy (SPT) visit most strongly indicates the need to return to active periodontal therapy?
Answer: A single site with 2 mm attachment loss since last visit
Progressive attachment loss at any site signals disease activity and warrants re-entry into active therapy.
For a high-risk periodontal maintenance patient, what is the most evidence-supported recall interval?
Answer: Every 3 months
Evidence supports 3-month SPT intervals for high-risk patients to prevent recurrence of periodontitis.
During reaccreditation, ABP diplomates are required to submit documentation demonstrating which of the following?
Answer: Completion of approved continuing education credits
ABP reaccreditation requires evidence of ongoing professional development through approved continuing education.
Which systemic condition is MOST associated with increased risk of periodontal disease recurrence during maintenance?
Answer: Poorly controlled diabetes mellitus
Poorly controlled diabetes impairs immune function and tissue healing, significantly increasing recurrence risk during SPT.
The Periodontal Risk Assessment (PRA) model uses all of the following parameters EXCEPT:
Answer: Patient age at diagnosis
The PRA model assesses BOP%, residual pockets, tooth loss, bone loss pattern, smoking, and systemic disease — not age at diagnosis.
During SPT, what is the PRIMARY goal of subgingival debridement at sites with residual pockets ≥5 mm?
Answer: Disrupt and reduce the subgingival biofilm to a subclinical threshold
SPT debridement aims to suppress the pathogenic biofilm below levels that trigger destructive immune responses.